What is Electromagnetic Hypersensitivity (EHS)?

General Information

 

This page is under construction

 

EM Facts Consultancy Brochure:  Wireless Networks (Wi-Fi)

http://mcs-america.org/EMFFactsConsultancyBrochureonWiFi.pdf

 

Havas.  Health Concerns Associated with Energy Efficient Lighting and Their Electromagnetic Emissions. 2008

http://weepinitiative.org/LINKEDDOCS/scientific/08_Havas_CFL_SCENIHR.pdf

 

Goldsworthy.  The Cell Phone and the Human Cell (PowerPoint).  2008. 
http://www.mcs-america.org/GoldsworthyTheCellphoneandtheCell.ppt  

Goldsworthy.  The Cell Phone and the Human Cell: The Calcium Story. 2008. 
http://www.mcs-america.org/TheCalciumStory2008.pdf  

Etiology (Causation)

 

The NO/ONOO- cycle is implicated by Pall as being a plausible etiology for Multiple Chemical Sensitivities (MCS), Fibromyalgia (FM), Chronic Fatigue Syndrome (CFS), Post-Traumatic Stress Disorder (PTSD), and Gulf War Syndrome.  Peroxynitrite (ONOO-) is oxidized from nitric oxide.  Excess peroxynitrite depletes energy stores, which is perceived to cause extreme fatigue (Pall, ND).  Of more interest to those who suffer from MCS is the fact that peroxynitrite breaks down the blood brain barrier and excess levels allow greater access to the brain (Pall, ND).  This greatly increases the effects of chemicals on the brain.  Essentially a non-MCS person has a barrier that protects the brain from damage from low-level chemical exposure, however a person who suffers from MCS has little or no barrier making the brain subject to increased damage and reactivity with minute exposures most people do not react to.  The key effect of nitric oxide (NO) is that it inhibits cytochrome P-450 activity and slows degradation of hydrophobic organic chemicals (Pall, ND).  This means that excess nitric oxide slows down the body’s natural detoxification processes leaving MCS patients subject to the effects of chemical exposure longer than non-sufferers.  Between a reduced blood-brain barrier and increased time to naturally detoxify the body, MCS patients are subject to permanent and long-term brain and nervous system damage which includes toxic encephalopathy.

 

“The only etiologic mechanism proposed for each of these is a vicious cycle mechanism involving elevated levels of nitric oxide and its oxidant product, peroxynitrite.  This cycle may be initiated by a variety of diverse short-term stressors, including viral organic solvent exposure, and exposure to three classes of pesticides, organophosphorus/carbamate pesticides, organochlorine pesticides and pyrethroid pesticides).  Each of these short-term stressors are known to be able to trigger responses that lead to increases in nitric oxide levels.  Indeed, other initiating short-term stressors, including a protozoan infection,  carbon monoxide exposure, thimerosal exposure and ciguatoxin exposure are also known or thought to act to increase nitric oxide levels, as well” (Pall, 2006).

Prevalence

Treatment

The following agents have been predicted to be useful by Dr. Pall and Dr. Ziem (Pall, 2006) in the Pall/Ziem protocol to down-regulate the NO/ONOO- cycle biochemistry:

 

References

Nebulized Inhaled Reduced Glutathione (RX Only)

Nebulized Inhaled Hyroxocobalamin (RX Only)

Mixed Natural Tocopherols

Buffered Vitamin C

Magnesium as Malate

Four Different Flavonoid Sources:

Ginkgo Biloba Extract, Cranberry Extract, Silymarin, & Bilberry Extract

Selenium as Selenium-Grown Yeast

Coenzyme Q10

Folic Acid

Carotenoids Including Lycopene, Lutein and Alpha-carotene

Alpha-Lipoic acid

Zinc (modest dose)

Manganese (low dose)

Copper (low dose)

Vitamin B6 in the Form of Pyridoxal Phosphate

Riboflavin 5’-Phosphate (FMN)

Betaine (Trimethylglycine)

Green Tea Extract

Acetyl L-Carnitine

 

For more information on treatments for MCS, see Medical Treatment.

Disclaimer

This site is for informational purposes and is not intended to replace the examination, diagnosis and treatment of a licensed physician and no such claims are inferred.  MCS America will not be responsible for misuse of this information or the misuse of any information provided by it’s member organizations.  Articles, citations, links and information are not necessarily the opinion of MCS America and printing does not constitute MCS America’s endorsement.

 

 

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